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FCAR/CD89 Antibody (A59) - BSA Free

Novus Biologicals, part of Bio-Techne | Catalog # NBP3-09118

Clone A59 was used by HLDA to establish CD designation.
Novus Biologicals, part of Bio-Techne
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NBP3-09118

Key Product Details

Species Reactivity

Human, Primate

Applications

Flow Cytometry

Label

Unconjugated

Antibody Source

Monoclonal Mouse IgG1 kappa Clone # A59

Format

BSA Free

Concentration

1 mg/ml

Product Specifications

Immunogen

Ag8.653 myeloma cells

Specificity

The mouse monoclonal antibody A59 recognizes an extracellular epitope of CD89, a 55-100 kDa glycoprotein serving as a receptor for IgA and expressed mainly on granulocytes, monocytes and macrophages.

Clonality

Monoclonal

Host

Mouse

Isotype

IgG1 kappa

Applications for FCAR/CD89 Antibody (A59) - BSA Free

Application
Recommended Usage

Flow Cytometry

1-4 ug/ml

Formulation, Preparation, and Storage

Purification

Protein A purified

Formulation

PBS

Format

BSA Free

Preservative

0.02% Sodium Azide

Concentration

1 mg/ml

Shipping

The product is shipped with polar packs. Upon receipt, store it immediately at the temperature recommended below.

Stability & Storage

Store at 4C short term. Aliquot and store at -20C long term. Avoid freeze-thaw cycles.

Background: FCAR/CD89

The CD89 gene is a member of the immunoglobulin gene superfamily and encodes a receptor for the Fc region of IgA. Thereceptor is a transmembrane glycoprotein present on the surface of myeloid lineage cells such as neutrophils,monocytes, macrophages, and eosin

Long Name

IgA Fc Receptor

Alternate Names

CD89

Gene Symbol

FCAR

Additional FCAR/CD89 Products

Product Documents for FCAR/CD89 Antibody (A59) - BSA Free

Certificate of Analysis

To download a Certificate of Analysis, please enter a lot number in the search box below.

Product Specific Notices for FCAR/CD89 Antibody (A59) - BSA Free

This product is for research use only and is not approved for use in humans or in clinical diagnosis. Primary Antibodies are guaranteed for 1 year from date of receipt.

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